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Vanin-1 pantetheinase drives smooth muscle cell activation in post-arterial injury neointimal hyperplasia.
Dammanahalli KJ, Stevens S, Terkeltaub R. Dammanahalli KJ, et al. PLoS One. 2012;7(6):e39106. doi: 10.1371/journal.pone.0039106. Epub 2012 Jun 13. PLoS One. 2012. PMID: 22720042 Free PMC article.
Vascular smooth muscle cells (SMCs) contribute to neointimal hyperplasia in response to injury, by multiple mechanisms including modulation of oxidative stress and PPARgamma. ...We conclude that vanin-1, via dual modulation of GSH and PPARgamma, critically regulates the ac …
Vascular smooth muscle cells (SMCs) contribute to neointimal hyperplasia in response to injury, by multiple mechanisms including modu …
Vanin-1 inactivation antagonizes the development of adrenocortical neoplasia in Sf-1 transgenic mice.
Latre de Late P, El Wakil A, Jarjat M, de Krijger RR, Heckert LL, Naquet P, Lalli E. Latre de Late P, et al. Endocrinology. 2014 Jul;155(7):2349-54. doi: 10.1210/en.2014-1088. Epub 2014 Apr 8. Endocrinology. 2014. PMID: 24712878
Sf-1 transgenic mice developed adrenocortical neoplastic lesions (both dysplastic and nodular) with a frequency increasing with age. Genetic ablation of the Vnn1 gene in Sf-1 transgenic mice significantly reduced the severity of neoplastic lesions in the adrenal cortex. Th …
Sf-1 transgenic mice developed adrenocortical neoplastic lesions (both dysplastic and nodular) with a frequency increasing with age. Genetic …